By George S. Jr. Everly, Jeffrey M. Lating
This up-to-date version covers a number new themes, together with pressure and the immune process, post-traumatic tension and difficulty intervention, Eye move Desensitization and Reprocessing (EMDR), serious Incident tension Debriefing (CISD), situation administration Briefings in accordance with mass mess ups and terrorism, severe Incident pressure administration (CISM), spirituality and faith as pressure administration instruments, nutritional elements and pressure, and up to date info on psychopharmacologic intervention within the human rigidity reaction. it's a complete and obtainable consultant for college kids, practitioners, and researchers within the fields of psychology, psychiatry, medication, nursing, social paintings, and public health and wellbeing.
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Extra info for A Clinical Guide to the Treatment of the Human Stress Response 2nd Edition (Springer Series on Stress and Coping)
ANATOMY AND PHYSIOLOGY OF THE HUMAN STRESS RESPONSE 31 The effects of neural activation via the sympathetic system are those of generalized arousal within the end organs—what Hess (1957) referred to as an “ergotropic” response. The effects of activation via the parasympathetic system are inhibition, slowing, and “restorative” functions—what Hess called a “trophotropic” response. 3 (see Ganong, 1997). Although the most common form of neural autonomic stress responsiveness in human beings is in the form of the ergotropic response (Johnson & Spalding, 1974), simultaneous trophotropic responses have been observed in human beings as well (Gellhorn, 1969).
Arnold (1984) has written that “the sheer experience of things around us cannot lead to action unless they are appraised for their effect on us” (p. 125). She has hypothesized the anatomical locus of such appraisal to be the cingulate gyrus and the limbic–prefrontal neocortical interface (see Aggleton, 1992). Arnold (1984) notes that the granular cells of the limbic–prefrontal interface contain relay centers that connect all sensory, motor, and association areas. She states: These connections would enable the individual to appraise information from every modality: smells, via relays from the posterior orbital cortex; movement and movement impulses, via relays from frontal and prefrontal cortex; somatic sensations can provide data via relays from parietal association areas; and things seen could be appraised over relays from occipital association areas.
Although cortisol does exhibit some of these properties, aldosterone is about 1,000 times more potent as an electrolyte effector. As the prepotent mineralocorticoid, aldosterone may effect other physiological outcomes, among them increasing glycogen deposits in the liver and decreasing circulating eosinophils. Excessive activation of mineralocorticoid secretion in human beings has been implicated in the development of Cushing’s syndrome (hyperadrenocorticism) by Gifford and Gunderson (1970) and in high blood pressure and myocardial necrosis by Selye (1976).
A Clinical Guide to the Treatment of the Human Stress Response 2nd Edition (Springer Series on Stress and Coping) by George S. Jr. Everly, Jeffrey M. Lating